2 edition of Studies on the beta-adrenoceptor mediated facilitation of sympathetic neurotransmission. found in the catalog.
Studies on the beta-adrenoceptor mediated facilitation of sympathetic neurotransmission.
Carl Dahlo f
by AB Ha ssle, Dept. of Pharmacology, Research Laboratories, University of Go teberg, Dept. of Pharmacology in Go teberg
Written in English
|Series||Acta physiologica scandinavica, supplementum -- 500|
A H van den Meiracker, A J Man in 't Veld, H J van Eck, F Boomsma, M A Schalekamp, Hemodynamic and hormonal adaptations to beta-adrenoceptor blockade. A hour study of acebutolol, atenolol, pindolol, and propranolol in hypertensive patients., Circulation, /CIR, 78, 4, (), (). The aim of the study was to further explore the prejunctional β-adrenoceptor-mediated control mechanism of noradrenaline release from sympathetic nerves in response to preganglionic nerve stimulation (PNS) and local nerve stimulation of the portal vein, respectively, in the pithed rat. Baseline values as well as the increments of mean arterial blood pressure (Δ-BP), heart rate .
Dahlöf C. Studies on beta-adrenoceptor mediated facilitation of sympathetic neurotransmission. Acta Physiol Scand Suppl. ; – [Google Scholar] Dahlöf C, Kahan T, Ablad B. Prejunctional beta 2-adrenoreceptor blockade reduces nerve stimulation evoked release of endogenous noradrenaline in skeletal muscle in situ. Urethane produces a level of surgical anesthesia characterized by preservation of a number of cardiovascular reflexes. When the proper route of administration is used, and the use of unnecessarily high doses is avoided, urethane anesthesia appears to be suitable for a number of investigations at cardiovascular level. However in certain types of studies involving .
Beta-blockers antagonise the effects of sympathetic nerve stimulation or circulating catecholamines at beta-adrenoceptors which are widely distributed throughout body systems. Beta 1-receptors are predominant in the heart (and kidney) while beta 2-receptors are predominant in other organs such as the lung, peripheral blood vessels and skeletal. Abstract The aim of this study was to investigate angiotensin II (Ang II) receptor–, bradykinin receptor–, and β-adrenergic receptor–mediated modulation of norepinephrine release from human renal sympathetic nerves and to characterize the respective receptor subtypes cortical kidney slices were incubated with [3 H]norepinephrine, placed in .
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Acta Physiol Scand Suppl. ; Studies on beta-adrenoceptor mediated facilitation of sympathetic neurotransmission. Dahlöf by: Dahlöf C. Studies on beta-adrenoceptor mediated facilitation of sympathetic neurotransmission.
Acta Physiol Scand Suppl. ; – Enero MA, Saidman BQ. Possible feed-back inhibition of noradrenaline release by purine compounds. Naunyn Schmiedebergs Arch Pharmacol. Mar; (1)– Floras by: Get this from a library.
Studies on [beta]-adrenoceptor mediated facilitation of sympathetic neurotransmisson [sic]. [Carl Dahlöf]. Stj-5aarne L, Brundin J. Dual adreoceptor-mediated control of noradrenaline secretion from human vasoconstrictor nerves: facilitation by BETA-receptors and inhibitor by alpha-receptors.
Acta Physiol Scand. May; 94 (1)– Dahlöf C. Studies on beta-adrenoceptor mediated facilitation of sympathetic by: Studies on beta-adrenoceptor mediated facilitation of sympathetic neurotransmission. February Acta physiologica Scandinavica. Supplementum. Carl Dahlöf; Read more.
Request PDF | β2-Adrenoceptor-Mediated Prejunctional Facilitation and Postjunctional Inhibition of Sympathetic Neuroeffector Transmission in the Guinea Pig Vas Deferens | This study. Shortening of the stimulus train length to 10 pulses/ stimulation, so as to conserve neuronal NE, resulted in a significantly greater maximal facilitation as compared to that in experiments with 30 pulses (98 ± 17 vs.
67 ± 5%) (fig. 1), suggesting an inverse relationship between stimulus train length and prejunctional 13adrenoceptor-mediated. This study examines the role of prejunctional beta adrenoceptors in the modulation of sympathetic neurotransmission in the rat heart.
Under anesthesia, the heart with right cardiac sympathetic nerves attached was isolated and perfused with Krebs' bicarbonate buffer containing cocaine (10 microM), corticosterone (40 microM) and atropine (6 microM).
The present study was therefore undertaken to examine the effects of ANF on sympathetic neurotransmission in its reported primary target organ, the kidney. Methods Male Sprague-Dawley rats ( g) were anesthetized with sodium pentobarbital (55 mg/kg,ip) and the right kidney and renal artery were isolated.
Endothelium is an important regulator of vascular tone via release of various endothelium-derived substances. Several studies have reported that endothelium may decrease the release of noradrenaline from vascular postganglionic sympathetic nerves and thus neurogenic vasoconstriction.
Endothelium derived-mediators (adenosine and NO) can modify vascular sympathetic neurotransmission. The modulation of vascular adrenergic neurotransmission in the spontaneously hypertensive rat (SHR) and Wistar-Kyoto rat (WKY) mediated by alpha-adren.
Dahlöf C. Studies on beta-adrenoceptor mediated facilitation of sympathetic neurotransmission. Acta Physiol Scand Suppl. ; – Kupfer LE, Robinson RB, Bilezikian JP. Identification of alpha 1-adrenergic receptors in cultured rat myocardial cells with a new iodinated alpha 1-adrenergic antagonist, [I]IBE Circ Res.
Book. Jan ; George Paxinos Studies on beta-adrenoceptor mediated facilitation of sympathetic neurotransmission In the present study the response of the type I. Organized into seven parts, this book begins with an invited lecture on the kinetic analysis of the neuronal and extraneuronal uptake and metabolism of catecholamines.
Subsequent parts discuss the regulation of receptor-mediated events; presynaptic receptors in the peripheral and central nervous system; neurotransmitters; and receptor antibodies.
Nakamaru M, Jackson EK, Inagami T. Beta-adrenoceptor-mediated release of angiotensin II from mesenteric arteries. Am J Physiol. Jan; (1 Pt 2):H–H Rajanayagam MA, Musgrave IF, Rand MJ, Majewski H. Facilitation of noradrenaline release by isoprenaline is not mediated by angiotensin II in mouse atria and rat tail artery.
Potential involvement in beta-adrenoceptor-mediated facilitation of noradrenaline release. Naunyn Schmiedebergs Arch Pharmacol – PubMed Google Scholar Moreau N, Richer C, Vincent MP, Giudicelli JF (c) [Interaction between SRa new angiotensin II antagonist and sympathetic nervous system in pithed SHR rats].
Introduction. Previous studies have demonstrated that activation of prejunctional β‐adrenoceptors increases the stimulation‐induced release of noradrenaline (NA) from sympathetic nerves (see Langer, ; Majewski, ).In contrast, activation of prejunctional β‐adrenoceptors has been shown to decrease the electrically evoked release of endogenous adenosine 5′‐tripho‐sphate.
Discussion The aim of the present work was to study the relationship, if any, between the two control mechanisms claimed to regulate the amount of NA secreted from sympathetic neurons on arrival of the propagated nerve impulse: the hypothesis of PGE-mediated control and that of a-adrenoceptor mediated control, originally proposed by.
Inhibition of facilitation of sympathetic neurotransmission and angiotensin II-induced pressor effects in the pithed rat: Comparison between valsartan, candesartan, eprosartan and embusartan.
PDF | The present study was designed to characterize the neurogenic contraction of rat radial artery. Electrical field stimulation (EFS) evoked | Find, read and cite all the research you need. In conclusion, this study establishes for the first time that acetylcholine and carbachol can facilitate neurotransmission to the smooth muscle of the prostate from the guinea‐pig.
While it is unknown whether similar facilitation occurs with the human prostate, it is of interest that muscarinic M 1 receptors are likely to be involved in.Lakhlani PP, Eikenburg DC.
Dependence of prejunctional beta-adrenoceptor facilitation of sympathetic neurotransmission on stimulus train length and agonist exposure time. Eur J Pharmacol. Jan 5; (1)–Additional studies demonstrated that at infusion doses of AngII capable of decreasing body weight in rats, the in vitro effect of AngII to enhance sympathetic neurotransmission to ISBAT was increased (English and Cassis ) and rats exhibited increased oxygen consumption (Cassis et al.
). Collectively, these results suggest that a portion.